2 edition of Cholecystokinin and dopamine in relation to ingestional behaviour of rats found in the catalog.
Cholecystokinin and dopamine in relation to ingestional behaviour of rats
Hessah Abdul-Rahman Al-Naser
Thesis (Ph.D) - University of Birmingham, School of Psychology, 1994.
|Statement||by Hessah Abdul-Rahman Al-Naser.|
Food ingestion stimulates cholecystokinin (CCK) release from the proximal intestine, but the mechanisms involved are not well understood. To investigate this effect in vivo in intact rats, plasma CCK was measured after orogastric feeding of proteins, protein hydrolysates, amino acids, glucose, and starch. Effects of prolonged cholecystokinin administration on rat pituitary-adrenocortical axis: Role of the CCK receptor subtypes 1 and 2. Authors: Ludwik K. MalendowiczCited by:
CHOLECYSTOKININ (CCK) IS a peptide that is found in both the brain and the gastrointestinal tract. CCK is released from the duodenum and jejunum in response to the intraluminal presence of nutrient-digestive products, and it has been proposed to serve a feedback function in the short-term control of food by: Cholecystokinin, but not cholecystokinin-8 shows gastrointestinal site specificity in regulating feeding behaviors in male rats. Two separate experiments were performed to localize the gastrointestinal sites of action regulating meal size (MS), intermeal interval (IMI) length and satiety ratio (SR, IMI/MS) by cholecystokinin (CCK) 8 and
title = "In vitro response of rat gastrointestinal segments to cholecystokinin and bombesin", abstract = "In previous studies of the rat gastrointestinal (GI) tract, we have demonstrated specific binding of cholecystokinin (CCK) to the pylorus and of bombesin (BN) to the gastric fundus, gastric antrum, duodenum, and by: Bidirectional modulation of sIPSC frequency by CCK in dentate gyrus granule cells A, sIPSCs recorded from a granule cell before, during and after the application of CCK ( μ m).Application of bicuculline (10 μ m) at the end of the experiment completely blocked sIPSCs, suggesting that the recorded events were mediated by GABA A receptors. B, summarized time course of Cited by:
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Cholecystokinin (CCK) is an abundant and widely distributed neuropeptide that plays a modulatory role in a variety of behaviours. This paper focuses on the role of CCK in modulating anxiety-related and reward-related behaviours in key brain regions of the amygdala and mesolimbic dopamine system, by: Cholecystokinin (CCK) coexists with dopamine in a large proportion of the ventral tegmental and substantia nigra neurons in rodents primates.
In this review Jacki Crawley integrates the neurophysiological, behavioral, and release studies which demonstrate both excitatory effects of CCK, and facilitatory modulating effects of CCK on the inhibitory actions of dopamine, in the Cited by: Cholecystokinin (CCK or CCK-PZ; from Greek chole, "bile"; cysto, "sac"; kinin, "move"; hence, move the bile-sac (gallbladder)) is a peptide hormone of the gastrointestinal system responsible for stimulating the digestion of fat and ystokinin, officially called pancreozymin, is synthesized and secreted by enteroendocrine cells in the duodenum, the first segment of the External IDs: GeneCards: .
European Journal of Pharmacology, 91 () 77 Elsevier EFFECT OF CHOLECYSTOKININ ON SELF-STIMULATION BEHAVIOR IN RATS M,~TY,~S FEKETE I AXEL RENTZSCH 2, HELMUT SCHWARZBERG 2 and GYULA TELEGDY ~* i Department of Pathophysiology, University Medical School, Szeged, P.O.B.Hungary, and: Department Cited by: The aim of the present study was to examine the role of cholecystokinin (CCK) and/or cholecystokinin receptors subtypes (CCK1R and CCK2R) in the regulation of the thyroid gland structure and function.
Animals were autopsied after 6 days of treatment with CCK or CCK receptor-specific antagonists Cited by: 1. Abstract. The present study evaluated the effect of the selective and long-acting dopamine D 1 receptor antagonist SCH on several aspects of the ingestive behaviour of genetically selected alcohol-preferring rats, bred from Sardinian alcohol-preferring rats.
The effect of subchronic (8 days) subcutaneous drug administration was evaluated on the simultaneous Cited by: The cholecystokinin B receptor also known as CCKBR or CCK 2 is a protein that in humans is encoded by the CCKBR gene.
This gene encodes a G protein-coupled receptor for gastrin and cholecystokinin (CCK), regulatory peptides of the brain and gastrointestinal tract. This protein is a type B gastrin receptor, which has a high affinity for both sulfated and nonsulfated CCK Aliases: CCKBR, CCK-B, CCK2R. The data indicate that CCK acts through catecholaminergic mechanisms in the hypothalamus to influence feeding behaviour.
AB - Administration of cholecystokinin (CCK)( μg in a volume of 2 μl) into the lateral cerebral ventricle caused a decrease in intake of food but a relative increase in intake of water (or water-to-food ratio) in by: Cholecystokinin (CCK) has been implicated in stress and anxiety disorders.
We have studied the levels of different molecular forms of CCK and CCK receptor characteristics in rats kept for 1 h in individual cages and exposed to decapitation of conspecifics, and a control group which was decapitated immediately.
Total CCK concentration was found to be Cited by: "The neuropeptide cholecystokinin (CCK) has been shown to interact with dopamine in various ways, including attenuation of dopamine D1 receptor-mediated vacuous chewing and grooming. While we have demonstrated a clear role for the CCK(A) receptor in the attenuation of dopamine D1 agonist-induced vacuous chewing, studies of grooming yielded.
Cholecystokinin modulates mesolimbic dopaminergic influences on male rat copulatory behavior neurophysiological effects of dopamine. Since the mesolimbic dopamine system participates in the regulation of male rat sexual behavior, (DA) system emphasize this system's relation- ship to motivated or rewarding behaviors [5,25,44].
The. Regional brain cholecystokinin changes as a function of rough-and-tumble play behavior in adolescent rats Jeffrey Burgdorfa, Jaak Pankseppa,b,*, Margery C.
Beinfeldc, Roger A. Kroesb, Joseph R. Moskalb aJ.P. Scott Center for Neuroscience, Mind and Behavior, Department of Psychology, Bowling Green State University, Bowling Green, OHUSA. Research report Regional brain cholecystokinin changes as a function of friendly and aggressive social interactions in rats Jaak Pankseppa,b,*, Jeff Burgdorfa, Margery C.
Beinfeldc, Roger A. Kroesb, Joseph R. Moskalb aJ.P. Scott Center for Neuroscience, Mind and Behavior, Department of Psychology, Bowling Green State University, Bowling Green, OHUSA. Cholecystokinin and satiety in rats and rhesus monkeys Article (PDF Available) in American Journal of Clinical Nutrition 30(5) June with 56 Reads How we measure 'reads'.
Cholecystokinin Decreases Food Intake in Rats 1 Article (PDF Available) in Journal of Comparative and Physiological Psychology 84(3). Cholecystokinin reverses the β-endorphin-induced inhibition of maternal behavior in lactating rats when injected both intracerebroventricularly and into an important site for reproductive behaviors, the medial preoptic area (Felicio, Mann, & Bridges, ; Mann et al., ).
The blood-brain barrier permeabilities of the type-A cholecystokinin receptor antagonists devazepide and A (N(α)quinolinoyl-D-Glu-N,N-dipentylamide) have been compared with those of the reference compounds iodoantipyrine, which readily penetrates the blood-brain barrier, and mannitol, which does by: Rats are good subjects for experiments because of the extensive overlap in brain anatomy and function, as well as similar dopamine neuron systems.
Dopamine neurons within this midbrain area start in a part of the brain called the ventral tegmental Size: 1MB. The supradiaphragmatic afferent activity was recorded from the distal cut segment of a filament dissected from a bundle of the left cervical vagal nerve (n=8) in animals with total subdiaphragmatic ted cholecystokinin octapeptide (CCK‐8) were first dissolved in saline containing % bovine serum albumine (BSA) and diluted with by: Correct The rats ate the food whenever the light/buzzer was presented, even if they had just finished a meal.
In a series of experiments in rats, Weingarten et al. repeatedly paired a light/buzzer stimulus with the presentation of food over a period of 11 days. RESEARCH Open Access Cholecystokinin enhances visceral pain-related affective memory via vagal afferent pathway in rats Bing Cao1, Xu Zhang1, Ni Yan1, Shengliang Chen2 and Ying Li1* Abstract Background: Pain contains both sensory and affective by: Read "Regional brain cholecystokinin changes as a function of rough-and-tumble play behavior in adolescent rats, Peptides" on DeepDyve, the largest online rental service for scholarly research with thousands of academic publications available at your fingertips.Peripheral administration of the COOH-terminal octapeptide of cholecystokinin in doses from 1 to micrograms per kilogram of body weight ( to micrograms per rat) significantly antagonized tail pinch-induced eating in rats, an animal Cited by: